{"id":7507,"date":"2025-02-10T01:34:33","date_gmt":"2025-02-10T01:34:33","guid":{"rendered":"https:\/\/shareinfo24.com\/?p=7507"},"modified":"2025-02-10T01:34:33","modified_gmt":"2025-02-10T01:34:33","slug":"scientists-discover-a-way-to-stop-cancer-by-turning-on-a-kill-switch-in-the-body","status":"publish","type":"post","link":"https:\/\/shareinfo24.com\/?p=7507","title":{"rendered":"Scientists discover a way to stop cancer by turning on a \u201ckill switch\u201d in the body."},"content":{"rendered":"

Scientists at UC Davis have found a new way to fight cancer using a receptor called CD95, or Fas. This receptor, which was not widely used in cancer treatments before, can act as a \u201ckill switch\u201d to make cancer cells self-destruct. Their discovery, published in\u00a0Cell Death & Differentiation<\/em>, could lead to new and more effective treatments.<\/p>\n

CD95 receptors, also known as death receptors, help control cell self-destruction, a process called apoptosis. These receptors are on the surface of cells and, when triggered, start a chain reaction that leads to the cell dying. While they are important for keeping the body\u2019s cells balanced, these receptors could also be used in cancer treatment, which has not been fully studied until now.<\/p>\n

Jogender Tushir-Singh, an associate professor at ,UC Davis, and his team worked to understand how the Fas receptor could help treat cancer. Through their research, they found a part of the Fas receptor that causes cells to die. This discovery is an important step forward in cancer research, opening up new possibilities for treatment.<\/p>\n

\n

One major challenge in cancer treatment is when cancer cells become resistant to treatments like chemotherapy and radiation. Immunotherapies, such as CAR T-cell therapy, show promise but are less effective against solid tumors. The discovery of the Fas epitope could prevent cells from escaping treatment by causing them to self-destruct.<\/p>\n

<\/p>\n